Gaia Cecchia
Nutrition & Prevention, Italy
Title: Pathogenesis of alimentary diabetes: recovery by loss of 20% body weight, and by attainment of initial hunger as well as of low BG before meals
Biography
Biography: Gaia Cecchia
Abstract
Background & Objective: We attempted to train two diabetic adults as we suggested in the first abstract. Diabetic people are different from healthy people in this: they don’t develop any hunger sensation after meal suspension.
Methods: Training: recognizing Hinitial Hunger and associate this sensation to low Blood Glucose (76.6 ± 3.7 mg/dL). We tried to implement this training in two obese, diabetic adults.The two subjects consumed meals devoid of fats and carbohydrates (Very Low Energy Diet, VLED) for 6 to 12 months.
Results: At recruitment the two diabetic subjects (out of two) showed a BMI of 39 and 33 and they neither developed a BG decline to 76.6 ± 3.7 mg/dL nor any hunger sensation after 2-days eating suspension. Then Both subjects lost 13%-20% of their initial body weight; they recovered 76.6 ± 3.7 mg/dL of BG and hunger sensations before one – three meals a day, i.e.: they went out of diabetes.
Conclusion: Diabetes develops by inveterate conditioned intake (when previous energy intake has not been fully exhausted before meals), excessive fattening, excessive post-absorption emission of fatty acids from fatty tissues, permanent loss of BG decline to 76.6 ± 3.7 mg/dL and permanent loss of physiological signals of hunger. A healthy, non-diabetic life may be recovered by a painless loss of 20% body weight (No fats, no carbohydrates) and may be maintained by implementing IHMP at reappearance of hunger sensations. This means accurate energy intake planning instead of hunger endurance.
